Utilize este identificador para referenciar este registo: https://hdl.handle.net/1822/67150

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dc.contributor.authorCunha, Cristinapor
dc.contributor.authorGonçalves, Samuel M.por
dc.contributor.authorDuarte-Oliveira, Claudiopor
dc.contributor.authorLeite, Luispor
dc.contributor.authorLagrou, Katrienpor
dc.contributor.authorMarques, Antóniopor
dc.contributor.authorLupianez, Carmen B.por
dc.contributor.authorMesquita, Inêspor
dc.contributor.authorGaifem, Joanapor
dc.contributor.authorBarbosa, Ana Margaridapor
dc.contributor.authorVaz, Carlos Pinhopor
dc.contributor.authorBranca, Rosapor
dc.contributor.authorCampilho, Fernando-
dc.contributor.authorFreitas, Fatima-
dc.contributor.authorLigeiro, Dario-
dc.contributor.authorLass-Floerl, Cornelia-
dc.contributor.authorLoffler, Jurgen-
dc.contributor.authorJurado, Manuel-
dc.contributor.authorSaraiva, Margarida-
dc.contributor.authorKurzai, Oliver-
dc.contributor.authorRodrigues, Fernando-
dc.contributor.authorCastro, António G.-
dc.contributor.authorSilvestre, Ricardo-
dc.contributor.authorSainz, Juan-
dc.contributor.authorMaertens, Johan A.-
dc.contributor.authorTorrado, Egídio-
dc.contributor.authorJacobsen, Ilse D.-
dc.contributor.authorLacerda, Joao F.-
dc.contributor.authorCampos, Antonio, Jr.-
dc.contributor.authorCarvalho, Agostinho-
dc.date.accessioned2020-09-29T12:58:11Z-
dc.date.issued2017-
dc.identifier.issn0091-6749-
dc.identifier.urihttps://hdl.handle.net/1822/67150-
dc.description.abstract[Excerpt] To the Editor: Proinflammatory immune responses are critically required for antimicrobial host defenses; however, excessive inflammation has the potential to damage host tissues thereby paradoxically contributing to the progression of infection.[...]por
dc.description.sponsorshipSupported by the Northern Portugal Regional Operational Programme (NORTE 2020), under the Portugal 2020 Partnership Agreement, through the European Regional Development Fund (FEDER) (NORTE-01-0145-FEDER-000013), the Fundacao para a Ciencia e Tecnologia (FCT) (contracts IF/00735/2014 to A.C., IF/01390/2014 to E.T., IF/00021/2014 to R.S., and SFRH/BPD/96176/2013 to C.C.), the Conselho de Reitores das Universidades Portuguesas (CRUP), Portugal (Acoes Integradas Luso-Alemas A-43/16), the Deutscher Akademischer Austauschdienst (DAAD) (project-ID 57212690), the Fondo de Investigaciones Sanitarias (Madrid, Spain) (grant #PI12/02688) and the ERA-NET PathoGenoMics (grant #0315900A). The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.por
dc.language.isoengpor
dc.publisherMosby Inc.por
dc.rightsrestrictedAccesspor
dc.subjectAdultpor
dc.subjectAspergillosispor
dc.subjectFemalepor
dc.subjectHematopoietic Stem Cell Transplantationpor
dc.subjectHumanspor
dc.subjectInterleukin-10por
dc.subjectLeukocytes, Mononuclearpor
dc.subjectMacrophagespor
dc.subjectMalepor
dc.subjectPolymorphism, Single Nucleotidepor
dc.subjectYoung Adultpor
dc.subjectGenetic Predisposition to Diseasepor
dc.titleIL-10 overexpression predisposes to invasive aspergillosis by suppressing antifungal immunitypor
dc.typeletterToEditorpor
dc.peerreviewedyespor
oaire.citationStartPage867por
oaire.citationEndPage870por
oaire.citationIssue3por
oaire.citationVolume140por
dc.identifier.eissn1097-6825-
dc.identifier.doi10.1016/j.jaci.2017.02.034por
dc.date.embargo10000-01-01-
dc.identifier.pmid28389392por
dc.subject.wosScience & Technologypor
sdum.journalJournal of Allergy and Clinical Immunologypor
Aparece nas coleções:ICVS - Artigos em revistas internacionais / Papers in international journals

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